Acute aortic
regurgitation occur rapidly. Clinical features are different from chronic
aortic regurgitation.
In this session we will learn about
1. Etiology
of acute AR
2. Clinical
signs of acute AR
3. Difference
between acute AR and chronic AR
Acute AR etiology
1. Dissection
of aorta
2. Infective
endocarditis
3. Trauma
Features of acute AR
1. Pulse
rate à
Tachycardia
2. Increased
chance for hypotension
3. There
won’t be cardiomegaly
4. Increased
chance for pulmonary artery hypotension
-
Left parasternal heave
-
Loud P2 will be present
Following changes are noted in heart sound
- S1 is
soft
- S2 is
single and P2 is loud.
- LVS3, S4
are common.
S1 is soft because of the
elevated left ventricular end diastolic pressure closing the valve prematurely.
Murmur in acute AR
Soft EDM (early diastolic murmur) is
heard in acute AR.
It is due to low
cardiac output.There is decreased pressure gradient between aorta and left
ventricle due to elevated LVEDP.
In acute AR,
tachycardia will decrease the duration of diastole and obscure the murmur of AR.
Austin flint murmur
is produced due to premature valve closure due to elevated LVEDP.
Absent peripheral signs in acute AR is due to
peripheral vasoconstriction, which attenuate the peripheral signs.
How will you differentiate between acute and
chronic AR.
Acute
AR
|
Chronic
AR
|
|
Pulse
|
Pulses
parvus
|
Collapsing
pulse
|
Blood
pressure
|
Hypotension
|
Normal
BP
|
Apex
|
No
cardiomegaly
|
Cardiomegaly
present
|
Pulmonary
hypertension
|
Common
|
Uncommon
|
LVS4
|
Common
|
Rare
|
EDM(Early
Diastolic Murmur)
|
Short
EDM
|
Long
EDM
|
Severity of AR – Assessed with hills
sign