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Showing posts with label practical notes. Show all posts
Showing posts with label practical notes. Show all posts

Types of continuous murmur

 Continuous murmur with cyanosis

  • TOF with PDA
  • Pulmonary atresia with bronchopulmonary anastomoses
  • Pulmonary AVF
Continuous murmur with systolic > diastolic component
  • PDA
  • Peripheral Pulmonaryartery stenosis
  • Broncho pulmonary anastomoses
Continuous Murmurs with Diastolic Accentuation
  • Rupture of sinus of Vakalva (RSOV)
  • Coronary arteriovenous fistula
  • Anomalous origin of left coronary artery from pulmonary artery (ALCAPA)
  • Pulmonary arteriovenous fistula

Inspection for shape and movement of the chest

Looking from above (standing behind the patient), over the shoulders or the upper part of the chest.If standing or sitting is not possible for the patient, inspect the chest in Iying down position, patient lies absolutely straight in the bed in supine position) inspect from the

  • Top.
  • Foot end of the bed.
  • The sides in profile.
  • Head end.
  • Back (try to turn the patient to any one side).
The following are the points to note :
  1. Any deformity, fullness or depression (i.e. shape of the chest), apical impulse etc.
  2. Back (winging of the scapula, drooping of the shoulder, kyphoscoliosis, gibbus. skin changes).
  3. Whether both the sides of the chest arc moving simultaneously and symmetrically.
  4. Classically  winged scapula is found in paralysis of nerve to serratus anterior (C 6 ,7) and sometimes in facio-scapulo-humeral muscular dystrophy.
  5. Assessment of the expansion of the upper lobes is better achieved by inspection
  6. From behind the patient, looking down at the clavicles during moderate respiration.
  7. Equal on both sides - normal
  8. Reduced movement on one side -  pleural disease ,pulmonary disease
  9. Bilaterally reduced movement - in emphysema.

Causes of fourth heartsound

 LV S4  causes

  1. Systemic hypertension
  2. AS (left ventricular hypertrophy)
  3. LV myocardial infarction
RVS4  causes
  1. Pulmonary hypertension
  2. Pulmonary stenosis (Right Ventricular hypertrophy)
  3. RV myocardial infarction.
Features of RVS4
  1. Heard at LLSB
  2. Inspiratory augmentation present
  3. Associated with  a wave in JVP
  4. Seen in PAH and pulmonary stenosis
Triple rhythm
S1+S2+S3/S4

Quadruple rhythm
S1,S2 + S3 + S4.

Seen In:
  • Cardiomyopathy
  • Coronary artery disease
Summation gallop
S, S3 with merged S, & S4.

Causes of pathological S4
  1. Hypertrophic cardiomyopathy
  2. Systemic hypertension
  3. Coronary artery disease
  4. Myocardial infarction
  5. Ventricular aneurysm.
S3 -Ventricular distension sound.
S4 -Atrial contraction sound.

Achronym
LV : Left Ventricular
AS : Aortic Stenosis
RV : Right Ventricular

S1 - First Heart Sound
S2 - Second Heart Sound
S3 - Third Heart Sound
S4 - Fourth Heart Sound

Assessment of position of Trachea

Trail's sign

Shift of trachea produces prominence of sternal head of sternocleidomastoid on the side to which the trachea is shifted. It is called Trail's sign.

The pretracheal fascia encloses the clavicular head of stemomastoids muscle on both sides. When the trachea is shifted to one side, the pretracheal fascia covering the stemomastoid muscle on that side relaxes, producing the clavicular head more prominent on the side of tracheal deviation.

Causes of tracheal shift

Pleural disease - Shift to opposite side
  • Pleural effusion
  • Pneumothorax
Pulmonary disease-Shift to same side
  • Fibrosis and collapse of lung
Goiter - Shift of trachea to opposite side.

Position of the Trachea and Trail's sign



Syncope due to reflex affecting heart

Vasovagal syncope

It  is a very common cause of dizziness or syncope that  is characteristically seen in response to the following.
  • Fear
  • Sudden emotional stress
  • Anxiety
  • Physical or mental exhaustion
  • Pregnancy 
  • Anaemia. 
Vasovagal syncope is always preceded by warning symptoms such as nausea. weakness, sweating, epigastric discomfort, blurred vision, headache, tinnitus, difficulty  in concentrating, sighing and dizziness.

The heart rate decreases, and the patient appears pale. The syncope is transient,and  last a few seconds to a few minutes, and this may be prevented by immediately lying down. Rarely, this type of syncope can occur when the patient is recumbent.

Orthostatic hypotension
Orthostatic hypotension produces dizziness on arising or after prolonged standing and this can be related to reduction  in effective blood volume, autonomic nervous system dysfunction, or rarely, to circulating vasodilator substances.

Causes are 
  • Drugs-antihypertensive or antidepressant medications, vasodilators, and beta blockers
  • Diabetic autonomic neuropathy
  • Anaemia
  • Low blood volume
  • Large varicose veins
  • Pregnancy
  • Addison's disease (rare cause)
  • Secondary hypertension-pheochromocytoma.
Hypersensitive carotid sinus
It is suspected when the patient describes dizziness or syncope after hyperextension of the neck, turning of the head, or pressure over the area of the carotid sinus from a necktie or during shaving. The syncope is evanescent, with rapid and complete recovery.

Ataxic Gait (Cerebellar Lesion)

  • This gait is also called as reeling. staggering. drunken gait
  • This type of gait is seen in patients with cerebellar lesion and alcohol intoxication
  • The patient is ataxic and reels in any direction, including backwards and walks on a broad base.
  • The unsteady feet are planted widely apart and placed irregularly.
  • The steps are uncertain, some are shorter and some are longer than Intended, and the patient tends to fall or deviate to the side of cerebellar lesion.
  • The ataxia is equally severe whether the eyes are open or closed
  • The patient finds difficulty in executing tandem walking.
  • Gait ataxia is seen in lesion of upper vermis and anterior lobe of cerebellum
  • Titubant ataxia - ataxic gait with vertical oscillation of head and trunk. 

Anatomical peculiarity of facialnerve

Facial nerve is a Sensorimotor nerve with the following functions
  • Special visceral efferent (facial muscle)
  • General visceral efferent (submandibular, sublingual and lacrimal glands)
  • Special visceral afferent (taste from anterior two-thirds of tongue)
  • General somatic afferent (sensation from external auditory meatus, mastoid and pinna)
The upper half of the face has a bilateral representation ,whereas the lower half of the face has unilateral representation.
Nuclei of facial nerve are 4 in number
Motor nucleus contains dorsal and ventral group of cells and is situated in ventral pons
Superior Salivatory nucleus control salivation
Nucleus of tractus solitaries carry taste sensation from anterior 2/3 rd of tongue and sensation from external auditory meatus
Lacrimal nucleus. for lacrimation

There is two types of movements voluntary and reflexmovements
Volitional movement
There is supranuclear connection for volitional movement
It is unique for facial nerve
For Volitional movement the fibers start from  lower part of precentral gyrus , corticonuclear fibers descend to pons and cross to opposite facial nucleus. Nucleus  for the superior half of facial muscles receiving ipsilateral and contralateral supranuclear fibers. Nucleus  for the inferior half of facial muscles mainly receive contralateral supranuclear fibers .This explains why the upperhalf is spared in UMN lesion and only lower half is affected.
Variation
Occasionally lower half of face also has ipsilateral supranuclear innervation but it is less than contralateral innervation. This will result in only paresis of lower half in UMN lesion, if ipsilateral supranuclear innervation is also equal to contralateral  innervation both upper half and lower half may escape in UMN lesion.
Rarely upper half will have innervation predominantly from contralateral fibers. Thus in UMN lesion there is extension of weakness to upper half of face also.
For reflex movement 
Fibers from premotor area, extrapyramidal center, basal ganglia, through separate pathway, innervate the Nucleus from both sides but predominantly from Right cortex.
Lesion of this pathway produce Mimic facial palsy.
Nervus intermedius
It is the  Sensory counter part of facial nerve,it carry fibers of Superior Salivatory nucleus, lacrimal nucleus, and Nucleus of tractus solitarius.
It subserves the following
Somatic sensation of mastoid region ,part of pinnae, external auditory canal
Secretomotor fibers to lacrimal gland, salivary glands - sublingual and submandibular and
Visceral sensation – taste sensation from ant. 2/3rd of tongue.

Branches facial nerve

Branches at the Geniculate ganglion 
  • Greater superficial Petrosal nerve -supplies secretomotor fibers to lacrimal gland
Braches of Vertical mastoid segment
  • Nerve to stapedius
  • Chorda tympani -arise 5 mm above the stylomastoid foramen, carry taste sensation from anterior 2/3rd of tongue. It supplies secretomotor fibers to submandibular and sublingual gland
Branches at the level of Stylomastoid foramen
  • Posterior auricular braches-supplies occipitalis and auricular muscles
  • Digastric – Posterior belly of digastric
  • Stylohyoid supplies stylohyoid muscle
Branches in the Parotid region 
Temporofacial  branch
  • Temporal
  • Zygomatic
  • Upper buccal
Cervicofacial branch
  • Lower buccal
  • Mandibular
  • Cervical.
They supply muscles of face, scalp, and platysma.

Course of the facial nerve -the 7th cranial nerve

Key anatomical area you should remember in relation to anatomy of the facial nerve are the following
  • Pons
  • CerebelloPontine angle
  • Internal auditory meatus
  • Middle ear 
  • Stylomastoid foramen
Intrapontine segment 
Pons
Facial nuclei is situated in the pons
Sensory and parasympathetic fibers are carried by nervus intermedius
It curves around the 6th nerve nucleus to form facial collicullus and form the first genu around the 6th Cranial nucleus
Cerebellopontine  Angle
Nerve emerges at the ventrolateral portion of pontomedullary junction with Nervus intermedius and 8th nerve and lies in the cp angle
Meatal segment
Enters the internal auditory meatus with the 8th nerve with the nervus intermiedius in between
Labyrinthine segment
It  dip into the facial canal in the floor of meatal canal, reaches the medial part of tympanic cavity form the 2nd genu - geniculate ganglion – receives the Nervus intermedius.
It curves posteriorly at the genu giving the Greater superfical petrosal nerve at the genu
Then it travels backwards in the horizontal direction (tympanic segment is above the middle ear)
Mastoid segment
It turns back vertically downwards to emerge through stylomastoid foramen, then turns vertically in the vertical (mastoid) segment
It gives nerve to stapedius and chorda tympani nerve in the vertical part
Parotid region
The facial nerve emerge through the Stylomastoid Foramen and enters the parotid region
It emerges at the stylomastoid foramen
Leaves  the parotid gland by dividing to temperofacial and cervicofacial branches
finally divides into five terminal motor branches

What are the Signs and Symptoms of Cushing’s Syndrome

Cushing syndrome produce changes in body fat,skin,bone,muscle,resproductive system,cardiovasular and blood.
Changes in the body fat
  •          Body fat Weight gain
  •        Central obesity
  •          Rounded face
  •          Fat pad on back of neck (“buffalo hump”)
Changes in the Skin
  •  Facial plethora
  •          Thin and brittle skin
  •          Easy bruising,
  •          Broad and purple stretch marks,
  •          Acne
  • ·        Hirsutism
Changes in Bone
  •         Osteopenia
  •          Osteoporosis (vertebral fractures),
  •         Decreased linear growth in children
Changes in muscle
  •          Muscle Weakness
  •          Proximal myopathy (prominent
  • ·        Atrophy of gluteal and upper leg muscles
Changes Metabolism
  • Glucose intolerance/diabetes
  • Dyslipidemia
  • Changes in the Cardiovascular system
  • Hypertension
  • Hypokalemia,
  • Edema
  • Atherosclerosis 
Reproductive system changes
  • Decreased libido,  amenorrhea in women
Blood and immune system changes
  • Increased susceptibility to infections
  • Increased white blood cell count
  • Eosinopenia,
  • Hypercoagulation with increased risk of deep vein thrombosis and pulmonary embolism
Central nervous system changes
  • Irritability
  • Emotional lability
  • Depression,
  • Cognitive defect and paranoid psychosis

How to examine for Collapsing Pulse an OSCE guide

The term collapsing pulse is used to describe a pulse with a rapid upstroke and descent, and is characteristically described in aortic regurgitation.
Other  names of the collapsing pulse
  • Watson's water hammer pulse
  • Cannonball pulse 
  • Pulsus celer.
How to elicit collapsing pulse?
To elicit the collapsing pulse you have to palpate the carotids or the radial pulse.
For the radial pulse:
  • Ask the patient to fully pronate his forearm.
  • Place your right hand on the radial pulse.
  • Grasp the patient's forearm with left hand (with your palm on the flexor aspect of patient's forearm).
  • Raise the hand above the level of the patient's head
  • Repeat the manoeuvre to note the accentuation of the collapse in the elevated position.

How to examine for radiofemoral delay.an OSCE guide

Radiofemoral delay is an important clinical sign that help to detect the coarctation of aorta
How to elicit radiofemoral delay?
To detect the radiofemoral delay you have to palpate the radial and femoral artery simultaneously.Normally the time taken for the pulse wave to reach the radial artery after the cardiac systole is 80 milliseconds and for the femoral artery it is 75milleseconds.If the femoral pulse is delayed compared to radial pulse it is called as radiofemoral delay.
Causes of radiofemoral delay
Coarctation ol aorta
Atherosclerosis of aorta.
Thrombosis or embolism of aorta
 Aortoarteritis.



How to examine for Femoral pulse:an OSCE guide

The femoral pulse is palpated over the ventral thigh between the pubic symphysis and anterior superior iliac spine with the middle and index fingers.
How to examine for femoral pulse?
  • Ask the patient to lie supine,
  • Make the leg partially flexed: abduct and externally rotate the hip,
  • Feel the pulse below the midinguinal point.
Comment on the following
  • Rate, rhythm, character, volume.
  • Character of the vessel wall.
  • Palpability of all vessels.
  • Radio-femoral delay.
Clinical significance
Examination of peripheral pulse is imporatant for detection of radiofemoral delay

This is very important for students preparing for USMLE and MRCP

How to examine for carotid artery an OSCE guide

The common carotid artery is palpated on the neck below the jaw and lateral to the larynx/trachea (that is mid-point between your earlobe and chin) using the middle and index fingers.
It can be felt between the anterior border of the sternocleidomastoid muscle, above the hyoid bone and lateral to the thyroid cartilage
Precaution on palpating the carotid artery
  • The carotid artery should be palpated gently 
  • The patient should be in  sitting or lying down posture. 
  • Stimulating its baroreceptors with low palpitation can result in  severe bradycardia or even stop the heart in some sensitive persons. 
  • Two carotid arteries of a person should not be palpated at the same time. as it may limit the flow of blood to the head, possibly leading to fainting or brain ischemia.
How to examine for carotid pulse?
  • Ask the patient to look straight ahead
  • Use your thumb and press it backwards
  • The pulse is felt at the level of the medial border of the sternomastoid muscle and lateral to the thyroid cartilage.
Comment on the following
Rate, rhythm, character, volume.
Character of the vessel wall.
Palpability of all vessels,


This is very important for students preparing for USMLE and MRCP

How to examine for Brachial pulse:an OSCE guide

Brachial artery pulse is located on the inside of the upper arm near the elbow
The brachial artery is palpated on the anterior aspect of the elbow by gently pressing the artery against the underlying bone with the middle and index fingers. 
Clinical significance 
This pulse is commonly used to measure blood pressure with a stethoscope and sphygmomanometer
How to examine for brachial artery pulse?
  • Partially flex the elbow,
  • Feel the pulse over the elbow with thumb or fingers
Comment on the following
Rate, rhythm, character, volume.
Character of the vessel wall.
Palpability of all vessels,

How to examine for Radial pulse:an OSCE guide

Radial artery pulse is located on the lateral of the wrist , it can also be found in the anatomical snuff box
The radial pulse is palpated immediately above the wrist joint near the base of the thumb (i.e., common site), or in the anatomical snuff box (i.e., alternative site), by gently pressing the radial artery against the underlying bone with the middle and index fingers.
Clinical significance
The examination of radial pulse is very useful in the following condition
Radioradial delay-seen in thoracic inlet syndrome and takayasu disease
Radiofemoaral delay in Coarctation of aorta
How examine for radial pulse
  • Semipronate the forearm,
  • Flex the wrist
  • Feel the pulse near the wrist,
Comment on the following
Rate, rhythm, character, volume.
Character of the vessel wall.
Palpability of all vessels,
Radio-femoral delay.


This is very important for students preparing for USMLE and MRCP

Examination of skin lesions an OSCE guide

1. Use good lighting: preferably natural lighting.
2. Patient is adequately exposed.
3. Comment on:              
  • Type of the lesion—primary and sequential: colour.
  • Shape of the lesions.
  • Arrangement of two or more lesions.
  • Distribution of the lesions—examine specifically the hair, nails and mucous membranes.
4. Do palpation to confirm findings.
5 Tell the probable diagnosis.
6. Do or tell one specific test to clinch the diagnosis.
Note
1. Primary lesions They could lie
  • Flat (in the plane of the skin): Macule.
  • Elevated (above the plane of the skin): Papule, plaque, vesicle, bullae, pustule, cyst, wheal.
  • Depressed (below the plane of the skin): Ulcer, erosion.
2. Sequential lesions (occur over a primary lesion) Scaling, dry or wet exudation or llchenification.
3. Shape Round, oval, annular, iris, serpiginous, umbillicated. polygonal or polycyclic.
4. Arrangement herpctiform. zosteriform. reticular, linear or serpiginous.             '
5. Distribution Symmetrical or asymmetrical, exposed areas, intertriginous areas, sites of pressure, local or generalised.
6. Specific tests Include microscopic examination of the scales, crusts or exudate. Tzanck test. Auspltz sign. Nikolsky's sign. Koebner's phenomenon, patch test. etc.
7. The manifestations of a few common diseases are:
  • Iris lesion or target lesion: Erythema multlforme.
  • Annular lesions with scaling: Dermatophytosts. psoriasis and pityriasis rosea.
  • Annular lesions without scaling: SLE. Secondary syphilis.
  • Bilateral symmetrical eruptions: Drug or endogenous  hypersensitivity
  • On exposed areas: Pellagra, photosensitivity.
  • On interlriginous areas: Candidiasis.
  • On sites of pressure: Psoriasis.
  • Velvety brown pigmentation of the axilla and groins: Acanthosis nigricans.



This is very important for students preparing for USMLE and MRCP

How to elicit Pemberton's sign:an OSCE guide

Pempertons sign is seen in retrosternal goitre
  • The  patient raises both his/her arms until they touch the ears. 
  • Hold up for sometime. 
  • Look for congestion of the face, cyanosis and distress in a retrosternal goitre.

This sign demonstrate the presence of latent pressure in the thoracic inlet
Pemberton's sign was named after Dr. Hugh Pemberton
A positive Pemberton's sign is seen in  superior vena cava syndrome (SVC), as  a result of a mass in the mediastinum. the sign is most commonly described in patients with substernal goiters where the goiter “corks off” the thoracic inlet.this maneuver is very  useful in any patient with adenopathy, tumor, or fibrosis involving the mediastinum

How to elicit Joffroy's sign an OSCE guide

In Joffroy's sign there is a lack of wrinkling of the forehead when a patient looks up with the head bent forwards

Joffroys sign is a sign of hyperthyroidism.It is elicited as follows
  • Patient's face is inclined downwards.
  • Patient attempts to look up.
  • Look  for absence of wrinkles on the forehead.


How to elicit Von Graefe's sign:an OSCE guide

Von Graefes sign is an important eye sign in hyperthyroidism.It is elicited as follows
  • The patient looks straight ahead.
  • Ask the patient to look down.
  • Look for a lag of the upper eyelid.
It is the lagging of the upper eyelid on downward rotation of the eye and indicate  underlying  Graves' Disease
The terms lid lag and von Graefe's sign have been used interchangeably in the past; but, they are distinct signs of downgaze-related upper eyelid static position and dynamic movement, respectively.